1
in
30
plotted against age at 5 years post exposure, 1959. Solid
line represents mean values for comparison female population.
T
T
7
BETWEEN THRESHOLDS
24h
=
T
PERCENT CIFFERENTIAL OF RED CELLS
a
— MEAN OF 53 NORMAL
O=5 YEARS POST EXPOSURE
@7 UNEXPOSED POPULATION
AMERICANS AGE
>15
|
4
7
---- MEAN OF I? MARSHALLESE|~
> 15
Po a -- de.
CUMULATIVE
PERCENT
“% TOTAL COUNT
AGE
=7
37
L
4.1
|
4.5
L
4.9
.
\
3.3
15
37
REC «11078
20
25
3% 35
40 45 50 55
THRE SHOLO
60 65 70
75 890
Figure 45, Price-Jones sizing of red cells showing mean of
17 Marshallese curves (from both exposed and compari-
Figure 44. Cumulative distribution curve,
Rongelap red blood counts, 1959.
son groups) compared with mean curveof 53 Americans.
erythropoetic depression compared with the unex-
deficiency. The serum proteins and serum vitamin
posed levels, but not as distinct as indicated by
erythrocyte counts in 1959. It would appear froma
these findings that some bone marrow damage
persists at 5 years post exposure.
The Rongelap people generally have a slight
anemic tendency (very slightly microcytic) com-pared with Americans. The cause is unknown.
Apparently it is not due to iron deficiency, since
serum iron levels are generally normal. It may
possibly be based on another typeof nutritional
11859581
xX
B,, levels, however, tended to the high.
The continued high level of eosinophils is unexplained. It was not believed that the types of
intestinal parasites noted on a previous survey
could account for it.
Hematological Leukemia Survey
Differential counts showed no increase in immature leukocytes that would cause one to suspect
leukemia in either the 1959 or 1960 survey. Alkaline
Mee
a
36 b
T
40
Figure 43. Individual red blood counts of exposed females
Figure 42. Individual red blood counts of exposed males
plotted against age at 5 years post exposure, 1959. Solid
line represents mean values for comparison male population.
qT
30
AGE (¥R)
AGE (YR)
100
20
~~
L
30
CG
ey
l
#70
d
wee D
p
60
aa
RAG «IQ ®
30.
Loe
RAC «tu ®
L
40
30
RONGE _AP UNEXPOSED 1959