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RONGELAP UNEXPOSED !959

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RONGELAP UNEXPOSED 1959

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——

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r 0:5 YEARS POST EXPOSURE
@ = UNEXPOSED POPULATION

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80

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20

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— MEAN OF 53 NORMAL
AMERICANS AGE > 15
«--= MEAN OF 17 MARSHALLESE
AGE

> 15

tod

dle. 1. -d4,-—4

PERCENT
CUMULATIVE

T

PERCENT DIFFERENTIAL OF RED CELLS
BETWEEN THRESHOLDS

%e TOTAL COUNT

100

i

70

plotted against age at 5 years post exposure, 1959. Solid
line represents mean values for comparison female popujation.

264+
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t

60

Figure 43. Individual red blood counts of exposed females

890

Figure 42. Individual red blood counts of exposed males
plotted against age at 5 years post exposure, 1959. Solid
line represents mean values for comparison male population.
"

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1

50

AGE (YR)

AGE ( YR)

To

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40

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49

4.
53

s7

13

20

23

30 35 40 45 30 55
THRESHOLD

60 65 70

75 80

RBC «10°

Figure 44. Cumulative distribution curve,
Rongelap red blood counts, 1959.

erythropoetic depression compared with the unexposed levels, but not as distinct as indicated by
erythrocyte counts in 1959. It would appear from
these findings that some bone marrow damage
persists at 5 years post exposure.

The Rongelap people generally havea slight

anemic tendency(very slightly microcytic) com-

pared with Americans. The cause is unknown.
Apparently it is not due to iron deficiency, since
serum iron levels are generally normal. It may
possibly be based on anothertypeof nutritional

Figure 45. Price-Jonessizing of red ceils showing mean of
17 Marshallese curves (from both exposed and compari-

son groups) compared with mean curve of 53 Americans.

deficiency. The serum proteins and serum vitamin
B,, levels, however, tended to the high.

The continued high level of eosinophils is unexplained. It was not believed that the types of
intestinal parasites noted on a previous survey
could accountforit.
Hematological Leukemia Survey

Differential counts showed no increase in immature leukocytes that would cause one to suspect
leukemiain either the 1959 or 1960 survey. Alkaline

Select target paragraph3