ABSTRACT
Absorbed dose D is shown to be a comnostte vartable, the vroduct of
fraction of cells hit (1.) and

cells.

OD is suitadle

organ.

cancer

cto cells

toa radiation ans it

since 1.5 1.5, DB approximates
the cell as well as

in tres

only Iy varies with D so

therefore does not mean
be lethal.

Rather,

_

that

fraction of cells

(nduction

trun a Shagle cell,

results in a wide uistrivution or

the expected mean value,

the

toa those

However, with low-level exposure (LLE) to radiation ana its

delivery of energy

and

because,

the mean energy density in

consequent provabdllicty of

and

(ait size) Z

for use with high level (ELE)

resulting acute organ effects
closely enough

the rean “dose™

a

the

z,

{1s constant with exposure.

nit sices

=,

Thus, sith Lit,

the apparent proportionality between “zose™

transfocmed is wtisleadiag.

that any (cell)

it means

stocrastis

Tnis proportiznalizy

dose, xo matter hew saall,-sac

that, in the exposure of 4 copulation 2

individual organisms consisting of

the coustituent relevant cells,

there

is

a small probability of particle-cell intecactions which tirunsler enerzy.
The probability of a cell transforming and

initiating a cancer

can imly

_
:2

greater than zero if the hit size (“dose of energy") to te cell is larze
enough.

Otherwise stated,

biologfcal organization,
to

that cell

the “dose”

namely,

tis ef

rh

4a5se z

if
ane

tve.

{is

definea at the proser

the cell and not

the organ,
ae

The above lrecen

develop a drastically diiferent approach

3S

as
are

q

.

only 2 larl<

to evaluation of

present system: absorbed organ dose, LET, a sta.idard raciation,

sog7uys

7

Lt

erty

that holds promise of obviating any requirement fur the coaponents sf

dose equivalent and rem.

q

aftVEa

the

KEL =),

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