41
capacity. No correlation was seen. Phe explanation tor the higher B,, levels is not app :
time.
to the Amerindians, Mongolians, and Eskimos, '*
among whom some Diego postive people are
» found...
. *-m.
The followingSiaedaBoeistics of 3L0
Studies of Genetically inheritec 4 raits.
Blood Groups.
The laburatory analysis of blood groups was conducsed by Dr. L.N. Sigsaman.
and colleagues and reporgit) a3 foows. The results
of the 1958 and 1959 studies were compined,
Marshallese represent signi@adint differences trom
those of their casterneighbors(Poly nesians) and
SUgESE A relauonship,
* or
Indonesians,
A relatively hig
£
taken to avoid dupliggtion. The resules of this.
broader sampling, compared to findings e&Sim- ©
Ahigh N ger
4. Extremely eeeefrequc ney.
+. Total absenge.g
“antl eyo factors.
monset al. for the sanw@ area“™ and for the Polyk ,
Haptoglobidg and Fonsferrins.
makinga total of310.individual bloods. Cagewes
%
_.
ith#loutheas Asians and
The distribu-
tionof the haptugtobin types un the 176 Rongelapnesians“" are presented in Tables 27, ae tS
ese tested is shown in
we JO, Data on all indiData on ail individualsF
are givén
viduals tested may be und in Appendix 6. This
pendix 6. The lindings Hockey
Pollows
vee sastiple included some families with two or more
offspring, in these, all siblings But une were reABO system. “Phe bisgiffrequengy ofdpeigen
moved by randomselection to give a saniple of
is again demonstrated,in conteagt &
(Beahice
ies included, at
of B genes in the Polynesians. The
€ oka: 124 vedeyiguapleStiewhich the:&
gene in this area has been noted repeatedly.‘ In
most, parents and one child.Phe distribution of
the haptoglobin types in this group did noc ditter
the present series a single individual of group A.B
was contirmed. The x° vaiue in this system ts 5.18
significantly from that in the total group. In each
(p=0.15). The excess of AB persons (expected 8,
case, agreement with the Hardy-Weinberg preobserved | 4) contributes, the major part of the x°
dictions was good, suggesung: that the population
deviation.
:
was homogeneousfor this tr t,t, the two
2. MN system. The extremely low frequency.ol M
sera with no ha
gene has een noted in many studies of this popuHp' gene ts 0
lation andarea, in contrast to its high trequency
req uency of¢ ’
in Polynesians. ‘’he x° value in thissystemis 23.7
( p= 0.00F), whichis statisticallyggitalid. ‘The ¢x
lies in sae fiading of 25 M
cans whereasdi
globin either.
manysera only44
Li.Ga0uld be éxpgected. T husit appears that the
N gefie in the heterozygote escapes detecuap. W
weidyres corrected for, the result would ook
evert reater frequency of N gene.
3. Rh- Hrsystem.Che marked frequency of the R'
(han regingipel
wetis again demahsipated, hj
in any otherstudy. The‘failuke&
rh g@gative persons sug
Lat
Rh-bir Frequency Among Mania t
ne ee
y
—
Marshallese’
genotype of the heterozygous”r os
This isfursiger supported By cba
1
Phenoty pe percent
Kh, Rh,
YO.)
Kh, Rh,
49
Rh,ch
change i| n x value can be caused by a sing e individiual o “unusual” #rouping,
4, GARE
systems The failure to tind in this group
of 310 a single person with a Diego or Kell factor
is noteworthy. The Marshallese, Maoris,'! and
Polynesians are similar in this respect, in contrast
Rh
Rh,
Gene frequency
R'
42°07
Me
Oz?
4.0
Lafess, _ td
ur
ii
y50
Ae
ps
t
R-
wg
4
ats
R'
(40)
OU
007