SKIN LESIONS AND EPILATION The early symptomsof itching and burning
of the skin and eyes were probably due mainly to
skin irradiation from the fallout material.
However, the chemical nature of this material

may have contributed to the irritation. It has
been noted (12) that irritating chemicals applied during or shortly after irradiation en-

hance the effects of radiation.

The lack of prominence of an erythema was
notable, particularly in view of the severity of
someof the lesions that developed. Wilhelmy
(13) states that erythema only occurs when the
dose reaching the papillary layer exceeds a cer-

tain level. Perhaps due to the low energy of

the beta radiation the dose to the dermis was insufficient to evoke the response. On the other
hand, the darkness of the skin and the development of hyperpigmentation may have masked
an erythema. Microscopically, a superficial
hyperemia was not prominent.
Wirth and Raper (10) point out that they

were impressed in their studies on P ™ radiation

of the humanskin with the difficulty of distinguishing between true erythema and tanning,
particularly in the skin of brunette individuals.

It was unfortunate that color filters were not
available to aid in distinguishing an erythema

us suggested by Harris e¢ al. (14).
In general, the length of the latent period
before developmentof lesions of the skin is considered to be roughly inversely proportional
to the dose of radiation (15, 16). In the present series of cases the relatively long latent petiod is suggestive of 2 low dose of radiation.
Due to the wide spectrum of beta energies and
particulate distribution of radioactive material,
strict comparisons cannot be made with previous experience. However, the later developmentofless severe lesions in the Ailinginne and
Rongerik groups as contrasted with earlier developmentof more seriouslesions in the Rongelap groupis in keeping with a lower skin dose
in the former, and a higher skin dose in the
latter. It is of interest, however, that the latent

period was dependent to some extent on
anatomical location.

The foot lesions, which

were generally the most severe lesions encountered, had a longer Jatent period than did

37

the less severe lesions occurring elsewhere on
the body. It is Jogical to assume that the feet
received a higher dose of radiation because of
proximity to the ground and this may explain
the severity of these lesions. The lonyer laten-:
period (desipte higher dose of radiation) may
be reluted to thickness of the epidermis, differences in length of mitotic cycles or other
inherent characteristics of skin in different
arens of the body.
The histopathological changes noted, such as
destructive und atrophic changes of the epidermis, disturbances in keratinization, and
atrophy of hair follicles, when taken together
are consistent with radiation injury to the skin
(9, 12, 17, 18, 19, and 20). Severe injury to
the dermis and blood vessels was not observed.
The minimal dermal injury with severe epidermaiinjury is in keeping with the large component of Jow energy beta material present, resulting in absorption of the greater portionof

the energy in the epidermis.

Hyperpigmentation of injured areas was a
consistent finding in the Marshallese and the

American Negroes.

Pigmented lesions were

also observed to a lesser extent in the white
Americans. Such pronounced pigmentation is
not characteristic of the usual lesions xs described following exposure to beta or penetrating radiation, but may be more typical of
the response to ultra soft roentgen or “(Grenz
rays” (21).
There is no satisfactory explanation for the
darker dusky-gray color that appeared in some
of the skin lesions us healing progressed. Vascular changes or pigment aberrations might
have been responsible. The return to near normal in this pigmentation by 6 months showed
the transient nature of this change. The continued absence of pigmentation at the site of
the deeper foot lesions at 6 months and 1 year
later suggests that the pigment-producing elements in these ureas were permanently damaged.

The unique features of the lesions such as
the marked pigmentation, the absence of obvious multiphasic response, the long latent period,

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