Fable 3
Mortalitv
Exposed
Year
No.
1996
1997)
25
38
& sx
44M
«76M.
1958
4]
$5M
1959
962
[962
1968200
1962
1964
N65
62
3
46
2b
46
52
057
66OF
nF
%YtM
21M
73 F
455 F
lO? F
Unexpenrd
Probable cause
Hypertensive heart disease
Coronary heart dpcase. diabetes
Acute varicella
Ovarian cancer
Cancer of cervix*
Arterionclerotic heart disease
Brain damage following fal! from tree
Fractured vertebrae
Pohomyelitis, bulbar
“Old age?)
Probable cause
1958
1999
1960
1960
1960
1962
1962
1965
857
854
935
927)
«861
9535)
848
BBB
65M
i5F
36M
«65M
OB F
48M
#41F
34M
Cerebral thromboum?)
Infection urmarv tract, diabetes
Pneumonia secondary to influenza
Preumons secondary to influenza
Diabetes, cancer cervix{ >)
Scatus asthmaticus
Neurasyphilis(?)
Asthmai?)
*Not confirmed by autopsy or hiopsy.
The birth rate for the past year was calculated
as ip the previous surveys from the number of
births per woman of childbearing age (15 tw 45
years). There vere 2.) such women in the exposed
¥group and 39 in the unexposed group. (Not in-
cluded in ether group were 4 unexposed women
whose spouses were exposed niales.) For the J-vecar
period in the exposed group 8 babies were born.
giving an average ol 0.17 births per wumanper
year, in the unexposed group 10 babtes were bom,
giving a shghily lower birth rate per woman 1U 135
per vear). The births wereall full-term normal deliveries, except one case as noted below.
Areview of the enure menstrual and obstetrical
history of the women cexamined in l4b4: in the
exposed and control groups iw gwenin Table 4. In
20 exposed women there had been a total of 4d
pregnancies, LY women delivering [15 living
children tor an average of 4.8 babies per woman
tor the 24 women i the group. The same lecun-
dit. was noted in the control women, 32 of the $9
women hasing been pregnant 203 ames and deTiweringe PSY living babies. averaging ¢.6 children
peromother The tistones of the age of onset of
Menstruation and development of menarche were
not too reliable, but the ages of onset for these
events “opear to be about the samein the exposed
and the control women
Table 5 lists the births and feral deaths by vear
since 1934 of Rongelap people. Since it was un-
certain whether the dist of births on Utrik Atoll
3
38
obtained during the 1963 survey was complete,it
was not possible to calculate an accurate birth rate
tor that group. However, the birth rate secmed tu
be about the same as nuted i. other Marshall
Island pepulauons.
Congenital Anomalies
A full-term suilbirth with congenital anomalies
cectromelus) was born to exposed parents in 1462.
‘This anomalyis not vers uncommon, and in view
of the statistical evaluation the question of radiation implication must be lett open, Que 24-vearvid exposed woman, Nu
4
6
bi
63
4
66
67
17
.
4
12
12
13
i3
7
14
71
Io
78
13
Sl
iJ
*
#4) was operated un tor
retopie pregnancy in L902 A case of congenital
heart detect had beer nered in a chiid born ofexposed parents several vears ago. This child died! at
4 months of age. Specttic genetic studies have not
been conducted on this relatively small population,
and only routine examination of new births has
been done. No unusual incidence of detecis has
been noted in the newborn Seme ot the detects
noted ro both exposed and unexposed chridren
inchide patent ductus arteriosus, congenital de-
formuty of the hip. and congenital hs popiasia of
the middle phalanx of the oth hnger
Miscorriages and Stillbirths
Except lor tne one ectopic pregnancy, no mis-
carriages were reported during the past 2-s.ear
period, One neonatal death (at 1 mouth of age)
due to intant diarrhea occurred in a twin born to
Ay
134
‘
Total subs.
(14)
j
TS
*Hvsterectomy. not im
EC
le