we
31
45
T
1
T
.
-
q
TT
}
MALES
PLATELETS x 107%
40h
.
4
|
|
we a py
jn
24 4 56 72 6 12 3 4 5 &
Oars
MO
YEARS
=
TIME
AFTER EXPOSURE.
PLATELETS x10°4
35
obb
08
Figure 43. Mean plateletvalues of exposed Rongelap
posure. Stars represent mean values of unexposed com-
4
|
4
|
.
@e
®
*
°
.
=
q
e
.
«
e
7
30
4
e
.
1O
20
4
°
iS -
{0
|
°
°
a
40
AGE (YR)
50
60
*
70
io
20
|
E
e
|
30.
40
.
”
.
AGE (YR)
PERIOD
COUNTS
50.
.
i
60
|
Ce
MINIMUM
(DAYS 26-30)
4
1
70.
=
{
4
80
—
f-
pf
60 -
/
40.
/
i;
80
Figure 45. Platelet counts
ae
i females
plotted against age. Solid, ine
cepires
unexposed female populatyey
-E
.
°
Bot
PERCENT
20+
20+
ico
CUMULATIVE
‘
4
4
[
25h
25
Figure 44. Platelet counts of exposed Rongelap males
plotted agalam age. Solid line represents meanlevel of
whatapeny he
unexposedmalepopulation.
!
i
30
4
-
FEMALES
al
|
|
2
parison population.
Tv.
4
sol
is
people from time of exposure through 7 years post.¢x-
PLATELETS x1o°*
|
°
.i }
8
,
f
/
f
/
f/f
1
f
f
7 YEARS POST EXPOSURE
- COMPARISON POPULATION -
961
/ /
16
24
32
40
PLATELETS x107*
48
56
64
7
Figure 46. Platelet cumulative percent distribution curves.
‘a
for exposed compared with unexposed population.
Children of Exposed Paresi
Peripheral blood elemempgiggf
chth
parents of:whom one or bot had been exposed
were compared with those of children with unexposed parents. These children were <7 years
=e
hevaciesduatt
hehco; aan
Noevidence of leukemia was detected in any of
the population studied based on leukocyte counts,
s we
-
differgntial counts, alkaline phosphatase stainitig
of leukocytes,gad counts of basophils in 400
vn No marked leukocytosis or immature
forms
ite cells. were observed. Relatively low
scorin
Aline phosphatase wesrecordedi.
some people, but in no case wasit associated with”
other findings suggestive of leukemia. Basophil
counts were elevated slightly (to 2.48%) in a 7year-old exposed male (No. 3) who had showed a
similar elevation in past surveys but no othersign
suggestive of leukemia. The individual values for