16
Table 7
Summary of Physical Findin . in Children, 1962
Control**
DhHOSL
WONnn
YVNnb Ow
NRF NON
ROKR OF hORON COO
Ooor-ovrnocod
Ww O&O
i ae
COOCCwW
Nonexposed of
exposed parents (37)
—
Swelling, preauricular soft tissues
Umbilical hernia
Born after
1954 (57)
Oe ODOCON TORK wv
Papilloma
Cheilosis
Sore on lip
Bizck spots—tongue
Denuded areas—tongue
Pigmentation—lips
Rheumatic heart disease
Systolic cardiac murmur (grade IT)
Born before
1954 (39)
to
Tinea versicolor
Tinea cruris
Vitiligo
Miliaria
Seborrhea—-scalp
Warts
t
CTF OM
Blood pressure taken
Hypertension
Acute otitis media
Chronic otitis media
Molluscum
OnoohOOCORK ON
Adenopathy
Palpable liver
Rhinitis
LA =o
Active skin lesions
OIOwW a
Exposed
(30)°
Hydrocele
Tromvocytopenia
Tracheotomy scar
Thoracotomy scar
Obesity
Pigeon breast
Contracture—fingers
1
1
*Number examined.
**A part of the group born after 1954 was used as the group “children of unexposed parents” to serve as a control for
“children of exposed parents.” Cf. footnote to Table 6.
growth spurt occurring earlier in girls than in boys
and the eventual superiority in size of boys at
maturity. In addition, with respect to stature, there
was a distinct tendency, among the boysonly, tor
those exposed at ages 1 through 5 to be shorter
than the unexposed boys of the same age (Figure
4). Although the differences ‘were statistically sig-
nificant only in the measurements at Ages 9, 11,
and 12, the retardation in stature of these boys
exposed at an carly age was apparent at all ages
at which measurements have been made. No tendency toward diminution in the magnitudes of the
differences was noted as the boys grew older. The
boys exposed at ages 6 through 8 showed no differences in stature from the control boys of the same
age. Since there were only two boys exposed in
the 12 through 13 age range, the data available did
not justify any conclusion regarding the effect of
exposure about the time of puberty.
The weights of the exposed and contral boys