Number of Spermatozoa (mm?)
lowering of motility and morphological abnor-
malities of spermatozoa were also observed.
The testicle of the fatal case, which died
206 days after the explosion, was remarkably
atrophic. possessing the interstitial tissue composed
of loosely arranged connective tissue.
Spermatogonia were greatly decreased in number,
and in some areas they were completelylacking.
No sperm was encountered within the lumen
of either seminiferous tubules or epididymis.
Indications of recovery were observed at about
Fig. 5.
5 and Table 2).
1954
1
|
,
1955
M (2) |
M
M
M
M
aa
(|
1957
1958
Changes in the Number of Spermatozoa
|
|
1959
1960
ff
1961 | 1962
|
1963 |
28
30
28
f
27
26
22
f
f
25
m
m
25
f
24
M
m
f
22
f
26
18
M
m
M
f
23
m
22
f
24
23
f
I
1
.
.
:
M (3):
M (2).
M.--e- Marriage
Meese Male
,,Age in
March 1954
27
38
(3)
(2)
(4)
(3)
M
13
+14
15
1959
Marriage and Childbirth of the Patients
1956 | 1957. 1958
A
1956
¢
M
M
|
f
f
M
m
f
A
if
18
22
39
|
Jenene No. of children at the time of exposure
ap venees Fatal case
farses Female
A. Abortus
m
27
20
RTaee
|
SPTa
1955
5. Liver Function
Slight disturbances of the liver function were
found in a few cases about 4 weeks after the
initial exposure. Later it became more obvious,
i.e. the repeated appearance of jaundice in several
cases, an increase of serum gamma-globulin con-
Most of them
began to reveal a sign of regeneration about 2
years after the exposure, and got children (Fie.
Date
0
l=
normal value in all the cases.
-—
1
1954
one year after the explosion in some patients,
but number of spermatozoa wasstill far below
Table 2.
3
spermia was found in [6 cases in November 1954,
e.g. about 8 months after the detonation. Both