spermia was found in 16 cases in November1954,
e.g. about 8 monthsafter the detonation. Both
lowering of motility and morphological abnor-
malities of spermatozoa were also observed.
The testicle of the fatal case, which died
906 days after the explosion, was remarkably
_atrophic, possessing the interstitial tissue composed
of loosely arranged connective tissue. Spermatogonia were greatly decreased in number,
and in some areas they were completely lacking.
No sperm was encountered within the lumen
of cither seminiferous tubules or epididymis.
Indications of recovery were observed at about
one year after the explosion in some patients,
but number of spermatozoa wasstill far below
normal value in all the cases. Most of them
began to reveal a sign of regeneration about 2
years after the exposure, and got children (Fig.
1
|
0
|
1 954.
Fig. 5.
‘1938
1956S
1958
1959
Changes in the Number of Spermatozoa
5. Liver Function
Slight disturbances of the liver function were
found in a few cases about 4 weeks after the
initial exposure. Later it became more obvious,
i.e. the repeated appearance of jaundice in several
cases, an increase of serum gamma-globulin con-
5 and Table 2).
Table 2, Marriage and Childbirth of the Patients
ae 1954
T-1
M (2)
5
6
M (4)
M (3)
2
3
7
1955
1956
1957
A
M (3)
M (2)
8
3
4
5
6
7
Mose Marriage
TI cooeee Male
f
Ml
om
(
f
22
M
25
m
m
m
f
Aceeees Abortus
24
22
26
18
m
m
23
22
24
23
f
m
f
oA
f
Jereeee No. of children at the time of exposure
fe oesee Female
25
M
f
M
f
!
{
M
f
M
|
|
f
M
f
m
97
f
M
m
M
30
28
26
M
!
,,Aee in,
M
m
M m
M (3)
M (2)
1963
¢
IM
12
1962
38
28
M
9
10
W
1961
27
m,
8
13
+14
15
16
1960
f
M
2
1959
f
M
K- |
1958
af renee Fatal case
m
18
22
39
27
20