16
Table 7
Summary of Physical Findings in Children, 1962
Contro!**
Swelling, preauricular soft tissues
Umbilical hernia
Hydrocele
Trombocytopenia
Tracheotomy scar
Thoracotomy scar
Obesity
Pigeon breast
Contracture—fingers
YONG
SoorowrnNoOcdod
NeENON
NMWOUMYA
NW oS
Pigmentation—lips
Rheumatic heart disease
Systolic cardiac murmur(grade II)
WhO A
Denuded areas—tongue
WNHNKOTWwW
Papilloma
Cheilosis
Sore onlip
Black spots—tongue
Orr OFF ORON DOO
Warts
—
Vitiligo
Militaria
Seborrhea—scalp
Nonexposed of
exposed parents (37)
DOT OCOwW
Tinea versicolor
Tinea cruris
Born after
1954 (57)
OroovnoceNVvNodre-N
Acute otitis media
Chronic otitis media
Molluscum
Born before
1954 (39)
nm OS
Blood pressure taken
Hypertension
OG W
Rhinitis
tv
CoO FR OU
Adenopathy
Palpable liver
(30)*
OnoOrfOO- O11
Active skin lesions
Exposed
tA ee
_
1
1
“Number examined.
** A part of the group born after 1954 was used as the group “children of unexposed parents” to serve as a control for
“children of exposed parents.” Cf. footnote to Table 6.
growth spurt occurringearlier in girls than in boys
and the eventual superiority in size of boys at
maturity. In addition, with respect to stature, there
was a distinct tendency, among the boys only, for
those exposed at ages | through S$ to be shorter
than the unexposed boys of the same age (Figure
4). Although the differences were statistically significant only in the measurements at ages 9, 11,
and 12, the retardation in stature of these boys
exposed at. an early age was apparent at all ages
at which measurements have been made. Notendency toward diminution in the magnitudes of the
differences was noted as the boys grew older. The
boys exposed at ages 6 through 8 showednodifferences in stature from the control boys of the same
age. Since there were only two boys exposed in
the 12 through 13 age range, the data available did
not justify any conclusion regarding the effect of
exposure about the time of puberty.
The weights of the exposed and control bovs