BRIEF REPORTS

716
gauge the effect of alcohol or diet on this difference.
Additionally, there is considerable interisland travel,
which makes it more difficult to isolate specific environmental factors that might account for the differ-

ence. As expected, no significant difference was noted
among the 3 groups classified according to radiation
exposure.

In the Framingham study (15), the prevalences

of gout at mean ages of 44 and 58 were 0.2% and 1.5%,

respectively, with the minimum ageon entry into the

study being 30 years. Applying the same minimum age

cut-off to the Marshallese at the time of SUA testing,
the prevalence of gout was 0.85% at a meanageof 50,
and 1.7% at 55. In this population, then, gout was
encountered about as commonly as in the Framingham
study.
It is not clear, at least from the present data,
that the Marshallese belong to the ‘‘one gouty family”
described by Kellgren for Pacific populations (16).
First, the prevalence and 5-year followup data on

clinical gout are not too different from those reported

in the U.S. Second, although mean SUA values were

high in the Marshallese, the cumulative frequencies of

values for men and for women 45 years of age or less
approach a normal distribution, indicating a higher
normal range than that found in the U.S. Hyperglobu-

linemia, often found in tropical areas, does not con-

tribute to the higher levels.
These findings may not accurately reflect the
true prevalence of hyperuricemia in the Marshall Islands because of the nonrandom nature of the investigation. Nevertheless, since inhabitants of the Marshall
Islands presently number about 32,000, our findings
describe more than 1% of that population.
Acknowledgment.

We gratefully acknowledge the

secretarial services of Mrs. Geraldine Callister.
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