In spite of differences in translocation pathways and biological half-times,
the radiation doses to critical organs are surprisingly similar for a given
intake situation. This leads us to use the Task Group model because it is
recognized by ICRP (ICRP-19, 1972) and the results using this model are not
too different from the more elaborate SDB and Modified models. Although still
the official model of ICRP, the ICRP-1E model is generally considered to be
outdated. The Task Group model was used to calculate the accumulated doses
and dose commitments (to 70 years) due to constant intake rates [A_ = 0.002 C
(pCi/day) and H_ = 0.19 C_
(pCi/day)], and the results are shown in Figures 8
and 9. Detailed tables of calculations based on 1 pCi/day ingestion or inhalation are given in Appendix A.

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PRACTICAL APPLICATIONS
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Our purpose in this discussion is to show how the results of a transport and
dose estimation model may be applied to the practical problem of deciding
whether and to what extent environmental decontamination might be required to
limit or reduce potential health hazards. The procedure suggested for this
purpose and outlined below is analogous to the procedure followed by ICRP in
calculating maximum permissible concentrations of radionuclides in air and
water. The principal steps involved are:
(1) fdentification of the critical
exposure pathway, (2) identification of the critical organ or organs, (3)
selection of maximum permissible dose criteria, (4) calculation of the corresponding maximum permissible concentration of Pu in soil (MPC) , and (5)
comparison of the (MPC) with estimated inventories of Pu in the surface soils
of contaminated areas at NTS.

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10-45
CRITICAL PATHWAY

The estimated plutonium ingestion rate for a hypothetical Standard Man living
in a contaminated area at NTS is about 100 times the estimated inhalation
rate; but, owing to the very small fraction of plutonium transferred from the
gastrointestinal tract to blood (3 x 10°°), che gastrointestinal tract is the
only organ that receives a significant dose from ingested plutonium. The
preferred dose estimation model based on ICRP recommendations (the Task Group
model, Figure 4) shows that inhalation accounts for 100 percent of the plutonium deposited in the Lungs and thoracic lymph nodes; and, for an ingestion/
inhalation ratio of 100, inhalation accounts for about 95 percent of the
plutonium in bone, liver, and kidney after 50 years of chronic exposure (Table 6).
Clearly, inhalation is the critical pathway.

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19-6

10,000

20,000
Days

FIGURE 8. PREDICTED CUMULATIVE DOSES DUE TO 239Pu IN
DIFFERENT ORGANS OF STANDARD MAN

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