26 somewhat greater in the exposed group in 1964 than in the unexposed comparison population. These abnormalities consist of polychromatic sheen, lenticular opacities of all degrees, and cataracts. The polychromatic sheen was noted as the earliest lens change and varied from a few fine granules in the earlier cases to large granular plaques in the more advancedcases. ‘These plaques were situated on the posterior lens capsule in the zone of specular reflection. The earliest cases showed yellowish granules which in some cases appearedslightly darker with a ‘“‘beaten brass” color. As the granules coalesce into a plaque, greenish and bluish hues appear — hence the name polychromatic sheen. Whetherthe polychromatic sheen seen following irradiation has unique and specific charac- teristics is still a debatable question. Some inves- Table 17 Residual “Beta Burns” Subject No. Age Sex Data Roughening and pigment variation on front of neck. Several pigmented macules ACF.* Perianal depigmen- 2 12. M_ 3 11 M_ 1! 60 M_ tation. systemic disease or intoxication. Such polychro- matic sheens were seen in 21% of the unirradiated Rongelap group and 26.5% of the exposed group. This difference is thought to be too small to implicate irradiation exposure with any degree of certainty, particularly in view ofthe slightly greater numberof older people in the exposed group. The incidence of lenticular opacities was also slightly greater in the exposed group (19%) than in the unexposed group (12%). Only one child (an 8-year-old female) complainedof defective night vision. This was thought to be due to vitamin A deficiency, since there were no pathological changesin the fundusofeithereye. Several years ago 12 children were encountered who hadgreatdifficulty in seeing at night. These children responded promptly with vitamin A treatment and dietary changes. Only two cases were noted with corneal pigmen- tation, previously seen in three cases (1962). This pigmentation was characterized bya fine, dark, linear streak of pigmentlying close to or on Bowman’s membranein the horizontal axis between the limbus and pupillary edge. It is believed that these changes may have been induced from beta radiation contaminating marginsofthe eyelids at the time of the accident. There were several findings which mayberesidual to the poliomyelitis epidemic of 1963: two Pigment changes left ACF, dorsum first right toe; pigmented nevi axilla. 17 13 F Scarring and pigmentation left ACF. 20 17 M_ Pigmented patch back of neck. 23 14 M_ Pigmented maculesleft axilla, front of neck and chest. Depigmented spots shaft penis. 24 23 =F Slight pigmentvariation on front of neck; several pigmented macules 34 55 F 39 25 F tigators contend that similar appearing changes can be detected in patients with retinitis pigmentosa and the early stages of cataracts which might be a complication of endogenous ocular or Mottled pigmentation both axillae. Pigmented area behindleft ear. dorsumleft foot. Slight roughening and pigmentation back of neck. Moles on front of neck. Slight roughening and pigmentation back of neck; pigment variations and slight hyperpigmentation dorsum right foot. 49 25 F 54 11 M 59 44 F Numerous pigmented macules both sides of neck and a few on arms and ACF. Mottled pigmentation and depigmentation on front of neck. Mottled pigmentation and depigmentation on back of neck. 63 46 F 64 40 F 65 il F Slight rugosity and pigmented ridges on back of neck. Mole back of neck; slight pigment variation and a few maculesfront of neck. Pigmentvariation and roughening front of neck. 67 24 75 22 78 $47 F Depigmented scars dorsum left foot. Slight pigmented area dorsum right first toe. | F Numerous pedunculated moles on sides and front of neck. 79 . 49 M_ Pigmented and depigmented scar posterior surfaceleft ear. *ACF=antecubitalfossa.