ERS?
;
NEEL ET AL.
264
RESULTS
i
Polymorphisms
Genetic polymorphisms were observed in six of the systems: haptoglobin, phosphoglucomutase-1, adenosine deaminase, acid phosphatase, 6-phosphoglucose de-
hydrogenase, and group specific component. Phenotype and allele frequencies are
given in table 2, Because of extensive inter-island population movement since the
TABLE 2
Grn FREQUENCIES For Stx GesEtic PotysronpHisiss Iw THE MARSHALL ISLANDS
PeENOTY?E
Adenosine deaminase ........
I
1974
1975
0
324
48
0
114
76
69
81
2
20
190
150
22
1974
1975
56
43
4
87
o31
53
99
181
84
1974
158
25
46
1
4
295
71
5
AEB
B
1974
1975
........ eewenes
Total ...cccccecccccesace
Phosphoglucomutase-It ......
1975
Total ...... cece eeee eae
137
A
Acid phosphatase ............
1974
1975
Total .... 2... ceca eens
6-Phosphogiuconate dehy-
drogenase ..........eceeeee
1974
1975
Total oo. ccc cece een cees
101
73
55
12
221
128
23
164
21
0
344
28
0
7
Gene Frequency
372
ADA! = 935
362
Gel = .732
364
Hpi = S21
371
FGMit == 891
372
ACPA = .766
372
6PGD4 = .962
.
11
120
180
Tora.
0
30
Total .........cceesceees
Haptoglobin*
18
2
137
. Total 2... 2. .eeefa eeeeeen
Group specific component ....
167
z-4
0
* The Hp® type was observed in four individuals,
t Two ezamples of the PGM phenotype 2-7 and 2 single phenotype 1-7 were observed.
Bikini incident, results are not presented separately by island. Varying numbers of
determinations for the systems listed in tables 1 and 2 are due to a considerable time
lapse between collection of the samples and their receipt in the laboratory. As a
consequence, either no pattern or an unclear pattern (even after repetition) was
occasionally obtained for a given trait. Three persons had the PGM; 7 phenotype.
Because of the occurrence of this variant in polymorphic proportions elsewhere in
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