——ne - 12 days, respectively. ais Fra fT BOTS i a ty ! a~ < Hg EA et : (7 ae His data show a lymph nodes burden of 0.40 pCi and a lung burden of 0.12 pCi for the year 1974. The Pu appearing in the urine from these two compartments during the next year would be 0.013 pCi. The contribution to the urine from inhalation and ingestion of Pu during the current year would be 0.0002 pCi (see Table 7). Therefore, the expected annual excretion from previous body burdens and present ingestion pathways ts approximately 0.012 pCi compared with the conputed annual amount of 0.365 pci’. These results suggest several possible interpretations: @ The urine samples were contaminated at the time of collection, and the Pu concentrations are significantly below those reported, e The transfer coefficient across the gut for biologically complexed Pu is much higher than 3 x 107°,the value developed from animal studies with various Pu compounds . e The transfer to urine from the blood for ingested Pu is greater then 8%. -@ The directtransfer of Pu to blood’ from the upper respirator) | tract is greater than 1k. e The estimated intake values through food, water, and air are * Incorrect. .. | .@ Any combination of the above. Interestingly, the plutonium body distribution models 7,16,17 and the Bikini pathway data (Tables 6 and 7) show that the major fraction of Pu presently entering the urine is by ingestion. , The absolute quantityof Pu predicted to reach the urine as a result of this annual intake is 1.4 x 1073 pCi instead of the annual 3.65 pCi computed from the reported data’. There would, of course, be an additional contribution to urine Pu levels due to ‘ 4 I0097121— i’: ° eed =