26 somewhat greater in the exposed group in 1964 than in the unexposed comparison population. These abnormalities consist of polychromatic sheen, lenticular opacities ofall degrees, and cataracts. The polychromatic sheen was noted as the earliest lens change and varied from a few fine granules in the earlier cases to large granular plaques in the more advanced cases. These plaques were situated on the posterior lens capsule in the zone of specular reflection. The earliest cases showed yellowish granules which in some cases appeared slightly darker with a “beaten brass” color. As the granules coalesce into a plaque, greenish and bluish hues appear - hence the name poiychromatic sheen. Whetherthe polychromatic sheen seen following irradiation has unique and specific characteristics is still a debatable question. Some investigators contend that similar appearing changes can be detected in patients with retinitis pigmentosa and the early stages of cataracts which might be a complication of endogenous ocularor systemic disease or intoxication. Such polychro- matic sheens were seen in 21% of the unirradiated Rongelap group and 26.5% of the exposed group. This difference is thought to be too small to implicate irradiation exposure with any degree of certainty, particularly in view of the slightly greater numberof older people in the exposed group. The incidenceof lenticular opacities wasalso slightly greater in the exposed group (19%) than in the unexposed group (12%). Only one child (an 8-year-old female) complained of defective night vision, This was thought to be due to vitamin A deficiency, since there were no pathological changes in the fundusofeithereye. Several years ago 12 children were encountered who had greatdifficulty in seeing at night. These children responded promptly with vitamin A treatment and dietary changes. Only two cases were noted with corneal pigmentation, previously seen in three cases (1962). This pigmentation was characterized by fine, dark, linear streak of pigment lying close to or on Bow- man’s membranein the horizontal axis between the limbus and pupillary edge. It is believed that these changes may have been induced from beta radiation contaminating margins ofthe eyelids at the time of the accident. There were several findings which may bere- sidual to the poliomyelitis epidemic of 1963: two Table t7 Residual “Beta Burns” Subject No. Age Sex Data 2 12 M_ Roughening and pigment variation on front of neck. Several pigmented macules ACF.* Perianai depigmentation. 3 11 M Mottled pigmentation both axiliae. Pigmented area behind left ear. 11 60 M_ Pigment changes left ACF, dorsum first ight toe; pigmented nevi axilla. F Scarring and pigmentation left ACF. 17 13 20 17 M_ 23 14 M_ Pigmented macules left axilla, front 24 23 F Slight pigment variation on front of Pigmented patch back of neck. of neck and chest. Depigmented spocs shaft penis. neck; severai pigmented macules dorsum left foot. 34 35 F Slight roughening and pigmentation back of neck. Moles on frone of neck. 39 25 F Slight roughening and pigmentation back of neck; pigment variations and slight hyperpigmenration dorsum 49° 25 F 54 11 M 59 44 F 63 46 F Slight rugosity and pigmented ridges on back of neck. 64 40 F Mole back of neck: slight pigment variation and a few macules tront ot neck, 65 1 F Pigment variation and roughening front of neck. 67 24 F 73 22 78 47 FF 79 +9 M_ right foot. Numerous pigmented macules both sides of neck and a few on arms and ACF. Mottled pigmentation and depigmentation on front of neck. Mottled pigmentation and depig- mentation on back of neck. Depigmented scars dorsum left foot. Slight pigmented area dorsum right first roe, Numerous pedunculated moles on sides and front of neck. Pigmented and depigmented scar posterior surface ieft ear. *ACF =antecubital fossa.