16
Table 7
Summary of Physical Findings in Children, 1962
Control **
Swelling, preauricular soft tissues
Umbilical hernia
Hydrocele
Trombocytopenia
Tracheotomy scar
Thoracotomy scar
Obesity
Pigeon breast
YON &
CC OF OVP NOOO
NRF NON
SHOR
NOAM
tr oS
Black spots—tongue
Denuded areas—tongue
Pigmentation—lips
Rheumatic heart disease
Systolic cardiac murmur(grade IT)
VNhOwW
Sore onlip
BORK OH RPORONCOO
Papilloma
Cheilosis
Nonexposed of
exposed parents (37)
—_
Warts
1954 (57)
TOTTW
Seborrhea—scalp
Born after
OroocrenocrrenN
Tinea versicolor
Tinea cruris
Vitiligo
Miliaria
N
DOK Ot
Acute otitis media
Chronicotitis media
Molluscum
OnoorfF OTK ON
Blood pressure taken
Hypertension
—
Adenopathy
Palpable liver
Rhinitis
un
Active skin lesions
SOW oo
(30) *
Born before
1954 (39)
wm Oo
Exposed
1
1
Contracture—fingers
*Number examined.
** A part of the group born after 1954 was used as the group “children of unexposed parents” to serve as a control for
“children of exposed parents.” Cf. footnote to Table 6.
growth spurt occurring earlier in girls than in boys
and the eventual superiority in size of boys at
maturity. In addition, with respect to stature, there
was a distinct tendency, among the boys only, for
those exposed at ages 1 through 5 to be shorter
than the unexposed boys of the same age (Figure
4). Although the differences were statistically significant only in the measurements at ages 9, 11,
and 12, the retardation in stature of these boys
exposed at an early age was apparent at all ages
at which measurements have been made. Notendency toward diminution in the magnitudes of the
differences was noted as the boys grew older. The
boys exposed at ages 6 through 8 showed nodifferences in stature from the control boys of the same
age. Since there were only two boys exposed in
the 12 through 13 age range, the data available did
not justify any conclusion regarding the effect of
exposure about the time of puberty.
The weights of the exposed and control boys