72
Hematok
T. KUMATORI
order of 350R to ¢
and LALANNE, C. M.,
Kivicra, K., Ikepa, N., Krmura, K., Kawanisul, H. and Kimura, M. (1956): ibid.
I, 521.
logie,6, 561, (1963)
Korasta, Y., Kumatori, T. et al. (1955): Iryo, 9, 1.
Tonizing Radiation
Kumatonl, T. and Miyvosut, K. (1963): Diagnosis and Treatment of Radioactive
Poisoning, 253, International Atomic Energy Agency, Vienna.
Kcuvatoat, T., Isninara, T., Uepa, T. and Mryosui, K. (1965): Medical Survey of
ietic tissue, Int. A
(1967)).
We have compar
the curves of Yu
Japanese Exposed to Fallout Radiation in 1954—A Report after 10 Years,
cases, and here a¢
National Institute of Radiological Sciences, Chiba.
same difference, <
tion of these data
the difference bet:
Mrkamo, Y., Mryosut, K., SHimizu, K., Isutxawa, K., Kurtyama, S., Koyama, Y. and
Kusatont, T. (1956): Research in the Effects and Influences of the Nuclear
of Hematology, 1, 29, Pan-Pacific Press, Tokyo.
Mirosui, K. and Kumatort, T. (1964): Nihon Ketsuekigaku Zensho, 3, 660,
Wee
ge St Meaty
Mivoset, K, and Kumatoat, T. (1955): Acta Haematologica Japonica, 18, 379.
Miyosui, K. and Kumatoat, T. (1962): Proceedings of 8th International Congress
cases and the p:
patients was that
patients the dose
with a maximum °
minus 4 per cent,
of accidents the u
less, and of the o
=
Bomb Test Explosions, II, 1313, Japan Society for the Promotion of Science,
Tokyo.
Maruzen Co., Tokyo.
Dr. KuMATORI:
opinion. In my
DISCUSSION
damaxe, and its relationship to external or internal radiation exposure?
Dr. KuMatori (Japan) : Since this fatal
case was anemic and revealed bone
marruw aplasia, he received blood
transfusions. Therefore, serum hepatitis can’t be ignored. However, at
the same time the existence of radiation-incduced liver damage should be
considered.
At present it is difficult
to decide the cause of his liver damage.
We should consider the probability
of the radiation-induced liver damage,
because in some publications we can
see a similar histological picture of
the liver of a patient who wasirradi-
ated therapeutically.
Dr. Sanc
Kumatori,
something
dencies on
(Korea): I thank you
I would like to ask
about hemorrhagic
these cases, Would
Dr.
you
tenyou
tell me how long did hemorrhagic
tendencies continue after the irradia-
tion?
Dr. Kumatoai: In my cases the he-
morrhagic
tendencies
were not so
‘severe. The prolongation of the bleeding time was observed in a few cases.
You can see normal bleeding time in
normally 3 minutes, and in 1 or 2
severe cases the bleeding time at the
critical stage was as long as about 10
minutes. I think this came back to
normal at about 10 weeks or so after
the exposures,
though
I have no
correct data here.
Dr. Sanc: And how about the fibrinogen concentration ?
Dr. Kumatoni: Fibrinogen concentration was almost normal from the
beginning of the examination.
Dr. Tupiana (France): I would like
to ask Prof. Kumatoni if be has an
idea of how uniform was the dose
delivered to your fishermen. I ask
you this question because we have a
rather large experience of total-body
irradiation for mostly kidney transplants in human patients, and there
are 2 main differences between your
results and the one we have also.
The first one is that aplasia of the
blood occurs much earlier in
BOON, aaa?
comment on the nature of the liver
most cases. In our cases, we used
Duke's method. The bleeding time is
ia
Concerning
rats
Dr. Citrton (U.S.A.):
the one fatality, would you care to
our
patients, and the rigidity also occurs
much earlier. The 2nd one is that the
minimum number of leukocytes is
much smaller in our cases, being of
the order 1 to 200 leukocytes per
cubic millimeter, after a dose of the
3
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