16 Table 7 Summary of Physical Findin . in Children, 1962 Control** DhHOSL WONnn YVNnb Ow NRF NON ROKR OF hORON COO Ooor-ovrnocod Ww O&O i ae COOCCwW Nonexposed of exposed parents (37) — Swelling, preauricular soft tissues Umbilical hernia Born after 1954 (57) Oe ODOCON TORK wv Papilloma Cheilosis Sore on lip Bizck spots—tongue Denuded areas—tongue Pigmentation—lips Rheumatic heart disease Systolic cardiac murmur (grade IT) Born before 1954 (39) to Tinea versicolor Tinea cruris Vitiligo Miliaria Seborrhea—-scalp Warts t CTF OM Blood pressure taken Hypertension Acute otitis media Chronic otitis media Molluscum OnoohOOCORK ON Adenopathy Palpable liver Rhinitis LA =o Active skin lesions OIOwW a Exposed (30)° Hydrocele Tromvocytopenia Tracheotomy scar Thoracotomy scar Obesity Pigeon breast Contracture—fingers 1 1 *Number examined. **A part of the group born after 1954 was used as the group “children of unexposed parents” to serve as a control for “children of exposed parents.” Cf. footnote to Table 6. growth spurt occurring earlier in girls than in boys and the eventual superiority in size of boys at maturity. In addition, with respect to stature, there was a distinct tendency, among the boysonly, tor those exposed at ages 1 through 5 to be shorter than the unexposed boys of the same age (Figure 4). Although the differences ‘were statistically sig- nificant only in the measurements at Ages 9, 11, and 12, the retardation in stature of these boys exposed at an carly age was apparent at all ages at which measurements have been made. No tendency toward diminution in the magnitudes of the differences was noted as the boys grew older. The boys exposed at ages 6 through 8 showed no differences in stature from the control boys of the same age. Since there were only two boys exposed in the 12 through 13 age range, the data available did not justify any conclusion regarding the effect of exposure about the time of puberty. The weights of the exposed and contral boys