178

TRE SHORTER-TERM BIOLOGICAL HAZARDS OF A FALLOUT FIELD

METABOLIC STUDIES WITH STRONTIUM-90

Taste I

Thedistribution of carrier-free Syin the rhesus monkey after intravenous
administration,
35 uC Sr®; Tor
ed 135 wO Ca
Pate

veoldfernale ce

Sri

%, dose

Skull. Rete eee eo nw ee
Afandible.
§ capulae.
caries.
ee ween
A oe
in ones ..
ee
met
n iat. .
emora.....
Radii and fibuloe. .

Faeh animal received

Tony + Sayremale 242 days postin-

ep

Sig ash

% alose

ie ash

% STW

little value because of the introduction into the
cae

wee
- =
wee ee ee eee
ee
a

--

wane ee
:
wee eee ee wee

Patellae. - wee eee cee

ee ee eee

Cervical and thoracic vertebrae, 2...
Lumbar and eaudal vertebrae . 2.0.22...
Felvis.
rene

M
a
Soft tissue balanee_____

-

-

7.7.00
2. 76
1.77
29
- 25
2.00
2. 53
2.93
1. 47
2&8
4,37
1. 82

18

2. 56
4.48
4.50
296

10
- OR

Q. 206
- 264
- 204
» 290
+ 295
. 278
- 41
- 207
217
. 202
197
- 18h

27

.
.
«
-

224
258
293
106

- OOF
: 009

2. 96
1.31
1. 28
31
35
- 90
2.05
2.39
14
L 67
1. 56
1, 20

0. 068
3
+ 138
~ 135
- 125
- 106
-116
- 136
- 134
+148
-U1
122

-35

175

2. 80
874
Oy
127

+ OF
: ra

0. 50
. 57
- 62
- 57
49
- 63
. 60
- 52
48
» 65
39
: 42

“60

135
: 148
(*)
. O16

: 54
: §2
(*) . 49

oor

: at

® Arthritie (1. e, pelvis and lumber and caudal vertebran heavily caicified and fused).

‘Included with lumbar and caudal vertehrae.

Tasun IT
Spe content of biopsy samiples of caudal vertebrae from injected adult male and
female rhesus monkeys, the first
two offspring of an injected female, and one control female. Tnjected animals received
35 uC Sr% intravenously
Aninial

Rosy.
Stupe.

Estimated age at

__

-..2.)
--|

sampling

5 @yr -..
5.5-6 yr...
3.5 yr...
20mg... ..
10 mo... ...
3-3.5 yr.

_.|
. |
~-f
..|
-|
-|

Samples of milk were obtained from Rosy for
four successive days shortly after the birth of
her second infant. Table IL] shows the Sr
level in the milk, the daily fecal excretion rate at
that time, and the Sr® content of plasma 1
month later. Exceptfor the first sample taken
3 days posipartum, the milk concentration was
from three to four times that in the plasma.
The blood-count data have proved to he of

Days postinjection

Rource

” Tied . tat]

688.0... 2. 2 Hooper Fdn .. ..j
628.200...
-- --do ..
«|
OF LL... na
-| Commereial.... 2.
(98 for mother)... Rory and Stupe ..
(402 for mother). . fo... dow...
None-coutrol . .
Commercial,
.

Bodyae ‘

ke)

4,85
ibd
3.35
2.95
1.99
3.84

dpm

sie

vertebrae
x
x
x
x
x
x

Table IV gives the Sr” contentof the first two
infant monkeys 3 months after birth, and 8 days
after birth of the third as determined by in vivo
scintillation counting. The last two lines in
Table IT show the Sr® burdens calculated from
bone biopsy samples for the two older monkeys,

Willie and Betty, at 20 and 10 months of age,
respectively.

The Sr® burdens of these animals apparently
had no ill effect upon their growth rate, as
shown in Figure 5. The growth rates of the
three siblings wereveryclose to that reported by
Pickering et al. [26] for infant monkeys of this
species raised under similar conditions. During
GROWTH OF THREE RHESUS MONKEYS.
BORN TO S90 BEARING MOTHER

106
10¢
108
102
10?
10!

eae ae”

3.2
8.2
11g
3.6 + 107
3.7 x 107°
2x 107%

- © LC =2,22 x 10¢dnm; body burdenbased on estimatefrom Table I that Se concentration In eattdal vertebrae ts representative
of total skeletal Br:

reneeah content. of monkey hone (plus marrow) takenas 27.5%; percent body weight of hone
estimatedat 18%for infants and femates and 10% for

* Actual measured body hurden15 aC; error of estimate of this sort Is thus In the range of W%.

Sr” in Infant Monkeys

“an| Retimated 8boa

Ee teaee

2.95
5.95
1.53
5.5
8.3
2.35

eolony late in 1954 of a blood parasite similar to
Bartonella. Theoriginal infected animals were
destroyed, but Rosy and Stupe apparentlystill
remain carriers, and the parasite is now endemic
in the colony,

aeo)
DAYS OF AGE
Friaure §.—Growth of three rhesus monkeys bern ww
Srbearing muther,

179

the first 6 months to a year the blood counts of
all three infants were within normal limits.*
Samples of pooled urine and feces from Betty

at 14 and 140 days of age contained 260 dpm/

day and 38.6 dpm/day, indicating that the Sr”
acquired by placental transfer was eliminated
fairly rapidly. Nearly a year after birth (305
days), Willie, the oldest, was still exereting Sr®

in the feces at a relatively high level: 205 dpm/

day. Morc recent excretion samples from these
two animals have not yet been analyzed.
Onthe basis of the data in Table IV, and the
calculated body burdens shown in Table IT,

approximate half times for elimination of Sr®
during thefirst 10 months of life were calculated

for the (wo older infants: 195 days for Willie,

and 155 days for Betty.

Extrapolation back to

the timeof birth of these two infants provides
a rough estimate of the placental transfer of Sr®,

Io the first-born, Willie, placental transfer
accounted for about. 0.3 wC Sr™, or 3 percent. of
the mother’s retained dose; the retention by the

mother was calculated from her half-time value

of 315 days. Rosy received her Sr injection
halfway through the second trimester of her
first pregnancy. The exirapolated Sr™ content
for the second offspring, Betty, was slightly
more than 0.15 eC, 2.9 percent of the mother's
retained dose 402 days postinjection. Wher
Henry, the third offspring, was 8 days old, an
in vivo count was on the borderline of the sensitivity of the counting method, and the Sr®
burden was estimated at something less than
0.1 2C, or slightly more than the 3 percent calculated for the other (wo. Although the number of individuals was small, and the measure~
ments subject. to errors of about 15 percent,
placental transfer of a gradually declining
burden of Sr” can be estimated at something
close to 3 percent of that in the mother shortly
before the birth of the infant.
8 At age [4 mo Betty's red blood cell count dropped to less than one
mitlion. .4 few days later she succumbed, apparently as the result of infection with tha blood parasite mentioned above. Her skeleton 18 currently being processed for assay WU was infeetod at the same time

but responded lo a lieroic course af treatment with “Araten,"chloroquin

hydrochloride (Wintlirop-Stcarns), and massiveLnjectionsof liver, Iron,

and folic acid. His red cell count has remalnedat from 4.1 to 4.5 millon
for the past $ months, and he has continued to gain welght at an
apparently normalrate.

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