sibly related to pachiaat ion exposure, IV. Thyroid Findings A. EARLY FINOINGS Baauninations for possible thycoid alaornialities were al aportaal partof the progran from Uie be. winning, At the taae of die aceiderat bt vias pict considered likely that the dhiervid: had aeecived a siellisienet (hose al radiodending te cesalt in aloaortndilies. dao reirospeet this proved tu de quite wrong. since Cha ruid pnjuey and tls sequelae have Deen the most sertuus date result of the fillaut exe posure donde Marshidlese people. NV chronoloicul review of events leading to the development of thyroid wbnownalities follaws, Beginning several years alien oapostire it was meted that oof 19 childrea exposed at 10 years wage showed retardation of growth.’ "Phin was pardcularly notable in the boys exposed at <3 vears of age (Pigure 26). Vhe cause of this retarcda- lion was bot lainediately apparent, [twas recog. nized that thyroid hornone deficiencyfrom thyroid injury could result in such growth retardation, Plawever. examinations duriag this early period did m, Figure 27. Growth-retarded boys expowd at age | year (Ne. 3. deft. and Ne. 3), Ginenths after start of treatment at oue EL (Leet), bot reveal aay thyroid abnornaities, and the PLT levels in these children as well asin all Marshallese were dathe normal te high range. Phe growth re- tardation gradually becaune more apparent, and iat DR pee Ip t = uo - . Vu Height (co) ‘ oe Bag at age 125 cece e Cantal geenap Leb : ? Vigure 20. Statural growth io Rongclip dove expames Figure 20. Rone dvagenesis of heads of hummeri in subject Sod.) pical of tivpothyroid: disease ¢ 103}, 2 eee at <5 yearns af age, M8. 1007, wo 0 ae ge et thee POT 2 eee wt tee Uh pete ge ewe A ‘Vhese data also imay be useful asa base line, should genede changes appear ia later gonerauions, pas