72
Hematolog
T. KUMATORI
order of 350R to 40
and LALANNE, C. M., «
logie, 6, 561, (1963) a
Jonizing Radiation o
Kivicra, K., Tkepa, N., Kimura, K., Kawanisat, H. and Kimuna, M. (1956): ibid.
I, 521.
Korama, ¥., Kumtaront, T. et al. (1955): Iryo, 9, L.
Kusatoart, T. and Miyosxt, K. (1963): Diagnosis and Treatment of Radioactive
ietic tissue, Int. At.
(1967)).
Poisoning, 253, International Atomic Energy Agency, Vienna.
We have comparec
the curves of Yug
cases, and here aga
Kuvatoal, T., Isnindana, T., UEDa, T. and Mivosui, K. (1965): Medical Survey of
Japanese Exposed to Fallout Radiation in 1954—A Report after 10 Years,
National Institute of Radiological Sciences, Chiba.
same difference, an
Mixamo, ¥., MrrosHt, K., SHimizu, K., IsHixkawa, K., Kuntyama, S., Koyama, ¥. and
Kuvtatori, T. (1956): Research in the Effects and Influences of the Nuclear
Bomb Test Explosions, II, 1313, Japan Society for the Promotion of Science,
Tokyo.
Miyvosut, K. and Kumatoat, T. (1935): Acta Haematologica Japonica, 18, 379.
Mryvosui, K. and Kumatori, T. (1962): Proceedings of 8th International Congress
of Hematology, 1, 29, Pan-Pacific Press, Tokyo.
Mirosut, K. and Kumatori, T. (1964): Nihon Ketsuekigaku Zensho, 3, 660,
tion of these data *
the difference betw-
cases and the pw
patients was that
patients the dose \%
with a maximum vé
minus 4 per cent, ©
of accidents the un
less, and of the or
Maruzen Co., Tokyo.
Dr. Kumatoar: I
opinion.
DISCUSSION
Dr. Citeton (U.S.A.): Concerning
the one fatality, would you care to
comment on the nature of the liver
damaue. andits relationship to exter-
nal or internal radiation exposure?
Dr. KuMatori (Japan) : Since this fatal
case was anemic and revealed bone
marruw aplasia, he received blood
transfusions. Therefore, serum hepatitis can’t be ignored. However, at
most cases. In our cases, we used
Duke’s method. The bleeding timeis
normally 3 minutes, and in 1 or 2
severe cases the bleeding time at the
critical stage was as long as about 10
minutes.
I think this came back to
norma! at about 10 weeks or so after
the exposures, though
correct data here.
[
have
no
Dr. Sanc: And how about the fib-
the same time the existence of radia-
rinogen concentration?
to decide the cause of his liver damage.
ration was almost normal from the
beginning of the examination.
tion-induced liver damage should be
considered. At present it is difficult
We should consider the probability
of the radiation-induced liver damage,
because in some publications we can
Dr. Kumatori: Fibrinogen concent-
Dr. TusiaNa (France): I would like
Kumatori, I would like to ask you
to ask Prof. Kumartori if he has an
idea of how uniform was the dose
delivered to your fishermen. I ask
you this question because we have a
rather large experience of total-body
irradiation for mostly kidney transplants in human patients, and there
tell me how long did hemorrhagic
The first one is that aplasia of the
see a similar histological picture of
the liver of a patient who was irradi-
ated therapeutically.
Dr. Sanc (Korea): I thank you Dr.
something about hemorrhagic tendencies on these cases, Would you
tendencies continue after the irradiation?
Dr. Kumatort: In mycases the he-
morrhagic
tendencies
were
not
so
severe The prolongation of the bleeding time was observed ina fewcases.
You can see normal bleeding time in
are 2 main differences between your
results and the one we have also.
blood occurs much
earlier in
our
patients, and the rigidity also occurs
much earlier. The 2nd one is that the
minimum number of leukocytes is
much smaller in our cases, being of
the order 1 to 200 leukocytes per
cubic millimeter, after a dose of the
%
4
i
In my ¢