|
60
|
70
i
80
RBL« 1 ®
Figure +3. Individual red blood counts of exposed females
plotted against age at 5 vears post exposure, 1959. Solid
line represents mean values for comparison female popu-
AGE (YR)
lation.
Figure 42. Individual red blood counts of exposed males
plotted against age at 5 years post exposure, 1959. Solid
line represents mean values for comparison male population.
100
i
T
co
\
\
|
L
t
“eo TOTAL COUNT
37
i
45
Rac xio §
LL
49
!
33
‘5
37
T
T
T
|
~
4
'
l
T
-—— MEAN OF 53 NORMAL
AMERICANS AGE >15
7
---- MEAN OF I? MARSHALLESE|”
AGE > 15
@=UNEXPOSECD POPLLATION
PERCENT
T
PERCENT OIFFERENTIAL OF RED CELLS
BETWEEN THRESHOLOS
=
T
O=5 YEARS POST EXPOSURE
CUMULATIVE
9
20
25
3 335
1
40 45 50 55
THRESHOLD
i
a
=e
60 65 70
L
75 80
.
Figure 45. Price-Jones sizing of red cells showing mean of
17 Marshallese curves (from both exposed and comparison groups) compared with mean curve of 53 Americans.
Figure 44. Cumulative distribution curve,
Rongelap red blood counts, 1959.
deficiency. The serum proteins and serum vitamin
erythropoetic depression compared with the unexposed levels, but not as distinct as indicated by
erythrocyte counts in 1959. It would appear from,
these findings that some bone marrow damage
persists at 5 years post exposure.
The Rongelap people generally have a slight
anemic tendency (very slightly microcytic) com-
‘pared with Americans. The cause is unknown.
Apparently it is not due to iron deficiency, since
serum iron levels are generally normal. It may
possibly be based on another type of nutritional
*,
B,. levels, however, tended to the high.
The continued high level of eosinophils is unexplained. It was not believed that the types of
intestinal parasites noted on a previous survey
could account for it.
Hematological Leukemia Survey
Differential counts showed no increase in im-
mature leukocytes that would cause one to suspect
leukemia in either the 1959 or 1960 survey. Alkaline
Mel.
i
30
~~
!
40
ny a
f
30
one
{
Pd
ABC at ®
L
40
30
AGE (¥R)