12
Hematolo
T, KUMATORI
order of 350R to 4
and LALANNE, C. M.,
Kimura, K., Ikepa, N., Kimura, K., Kawanisut, H. and Kimura, M. (1956): ibid.
logie, 6, 561, (1963)
I, 521.
Koyama, Y., KuMaTori, T. et al. (1955): Iryo, 9, 1.
Tonizing Radiation«
Kumatoni, T. and Mryosui, K. (1963): Diagnosis and Treatment of Radioactive
. Poisoning, 253, International Atomic Energy Agency, Vienna.
ietic tissue, Int. A
(1967)).
We have compare
the curves of Yu
‘
Kumatori, T., IsHtnana, T:, UEDA, T. and Mryosui, K. (1965): Medical Survey o
Japanese Exposed to Fallout Radiation in 1954—A Report after 10 Years,
National Institute of Radiological Sciences, Chiba.
cases, and here ag
Mixamo, Y., Mryosut, K., Suimizu, K., IsHikawa, K., Kurryama, S., Koyama, Y. and
of Hematology, 1, 29, Pan-Pacific Press, Tokyo.
Miyrosui, K. and Kumatori, T. (1964): Nihon Ketsuekigaku Zensho, 3, 660,
Maruzen Co., Tokyo.
DISCUSSION
Concerning
most cases. In our cases, we used
Duxe’s method. The bleeding timeis
damage, andits relationship to exter-
severe cases the bleeding time at the
Dr. Kumatori (Japan) : Sincethis fatal
case was anemic and revealed bone
marrow aplasia, he received blood
minutes. I think this came back to
normal at about 10 weeks or so after
transfusions. Therefore, serum hepa-
titis can’t be ignored. However, at
the same time the existence of radiation-induced liver damage should be
considered. At present it is difficult
to decide the cause of his liver damage.
Weshould consider the probability
of the radiation-induced liver damage,
because in some publications we can
see a similar histological picture of
the liver of a patient who wasirradi-
the exposures, though I have no
correct data here.
Dr. Sanc: And how about the fib-
rinogen concentration?
Dr. Kumatori: Fibrinogen concent-
ration was almost normal from the
beginning of the examination.
Dr. Tusiana (France): I would like
something about hemorrhagic tendencies on these cases, Would you
tell me how long did hemorrhagic
tendencies continue after the irradiationP
Dr. Kumatoni: In my cases the hemorrhagic tendencies were not so
to ask Prof. Kumatorti if he has an
idea of how uniform was the dose
delivered to your fishermen. I ask
you this question because we have a
rather large experience of total-body
irradiation for mostly kidney transplants in human patients, and there
are 2 main differences between your
results and the one we have also.
The first one is that aplasia of the
blood occurs much earlier in our
patients, and the rigidity also occurs
much earlier. The 2nd one is that the
minimum number of leukocytes is
ing time was observed in a few cases.
You can see normal bleeding time in
the order 1 to 200 leukocytes per
cubic millimeter, after a dose of the
ated therapeutically.
Dr. Sanc (Korea): I thank you Dr.
Kumatori, I would like to ask you
severe. The prolongation of the bleed-
“ We.
critical stage was as. long as about 10
much smaller in our cases, being of
Adilsh apanas’ ts
nal or internal radiation exposure?
normally 3 minutes, and in 1 or 2
+
Dr. Currron (U.S.A.):
the one fatality, would you care to
comment on the nature of the liver
Fest
Miyvosut, K. and Kumatont, T. (1955): Acta Haematologica Japonica, 18, 379.
- Mryvosui, K. and Kumatonri, T. (1962): Proceedings of 8th International Congress
Lat 5 of ja Sd aya te 5 +
Bomb Test Explosions,I, 1313, Japan Society for the Promotion of Science,
Tokyo.
a
Kumaton, T. (1956): Research in the Effects and Influences of the Nuclear
same difference, 2
tion of these data
the difference bets
cases and the pt
patients was that
patients the dose
with a maximum ‘
minus 4 per cent,
of accidents the u
less, and of the o
Dr. KuMATORIL:
opinion. In my