+100 in permanent epilation and dryness of the skin. Such lesions are fertile cround for the later development of malignant chanze. resulting from beta Lesions of the skin radiation are less likely to result in chronic radia~ tion dermatitis than are the lesions produced by more penetrating radiation such ag are sometimes seen following Xeray or radium therapy. Malignant chanzes in the skin have “een reported in animals following beta radiation (Raper et al. Brues, Glucksman, Kolotsky, Shubik), but so far as the authors are aware, such chanses have not been reported in the human being. Though malignancy usually develops at the site of chronic gadiation dermatitis, as a resuit of repeated exposures to radiation, it may develop as a sequel to mild exposures with little chronic changes in the skin. It has been reported to occur in animals following a gingle exe posure to beta radiation with little or no chronic change in the skin. (Raper et al.) In view of the auperficial nature of most of the Marshallese lesions and the lew evidence of chronic effects in the skin, the likelihood cf skin cancer in this group seems diminished. Od HI@T PATHCLOGY CF BETA LESIONS. B8y and large, the histopatho- logical changes in the skin pnroduced by beta radiation are much the same as those preduced by gamma or X rays. Since histological changes induced by the latter radiations have been well documented (MacKee, Cipollaro and Montgomery; Warren; Bloom and Bioom; and Walbach, etc.), a detailed description of the changes induced by beta radiation will not be presented. A limited number of studies cf the histological changes in the skin of animals (Snider anc Raper; Moritz and Henriques) and in man (Lowe-Beer) frcem beta radiation have been rescrted. In general the chances produced by beta radiation are more superficial than those produced by more penetrating radiations with relatively much wreater damage to the epidermis toan to tit dermis. With fallout radiation