Dees HII
SO

340

RADIATION STANDARDS, INCLUDING FALLOUT

Since the time of the last hearings on fallout before this committee,

much attention has been centered on the hazards posed by the production of carbon 14 as a product of nuclear detonations. No exact appraisal of this hazard can be given at the present time, but it is clear
that estimates of genetic damage must be revised upward because of
the ready incorporation of carbon 14 into living tissues and into the
hereditary materials themselves, and because of the long half-life of
carbon 14.
The recently issued new edition of “The Effects of Nuclear Weapons” places the dosage of emitted beta radiation from carbon 14 as

equal to that from cesium 1387, although delivered at a very much

lower dose rate; and the genetic damage from transmutation of carbon
14 into nitrogen 14 as equal to that from the emitted beta radiation.
Isee no reason to differ with these estimates.
There are other recent findings which increase our estimates of
genetic damage done by atomic radiations. At the Oak Ridge National Laboratory, D. L. Lindsley has shownthat in the fruitfly many
lethal mutations remain undetected in the usual method of screening
for sex-linked recessive lethals. Estimates of the total number of
lethal mutations produced by a given dose thus need to be increased
by 20 percent. Findings such asthis one and the role of carbon 14 in
the production of genetic damage tend to offset the reduction in the
estimates of overall genetic damage which derive from the knowledge
that low dose rates, with which we must be mainly concerned, are much
less potent in producing mutations than the high dose rates commonly
used in the past in experimental investigations.
In conclusion, I think it might be worth while to say a word about
the genetic damage done by fallout from weapons tests or possible
nuclear attacks upon the United States. I do not feel it advisable now
to engage in the sort of “numbers game” that has been played in the
past. It seems important to me to emphasize the vast range of uncertainty embodied in such estimates, an uncertainty extending over two
orders of magnitude, or a hundredfold difference. Insofar as the
effects being compared are produced by radiation at a low dose rate and

are linearly proportional to dose, it is sufficient to keep in mind that

the average accumulated fallout dose from weapons tests through
1959 to persons in the United States is estimated to be 0.15 roentgen,
lifetime dose. (The new edition of “The Effects of Nuclear Weapons”
states the dose as 0.1 rem.)
From this figure, taking the higher one, resulting from some 92
megatonsof fission explosion, one can extrapolate to the fallout exposure for a similarly housed and unprotected population in the
event of 1,500, 3,000, 10,000 megatons, or larger attacks or wars. Depending upon the assumptionsasto the relative proportionsoffission
and fusion, one can calculate fallout doses to target nations rather
inexactly and to nontarget nations more exactly.
It is worth emphasizing that the calculated dose from stratospheric
fallout to a nontarget nation located in the Northern Hemisphere

in the zone of heaviest. deposition, between latitudes 30° and 60°,

amounts to about 10 to 12 roentgens in the case of a 20,000-megaton
war of which 50 to 60 percent mightbe fission. In other words, a
nuclear war of such magnitude as to suffice to devastate the United
States, the U.S.S.R., and all of Western Europe, would produce fallout that would only equal or slightly exceed in terms of its genetic

LORSRSEBAeebeaFategereiseRe Coreinea

Select target paragraph3